​The purpose of the Core Restore® program is to support efficient detoxification. This guide will help you understand how you can reduce the toxic burden in your body as well as outline a step-by-step program to promote healthy detoxification. Upon completion of this program, you will experience the many benefits of improved
biotransformation, such as improved energy, better sleep, increased mental clarity and newfound vitality.
After you complete the 7-day detoxification program, you and your health care provider will assess your progress. He or she may recommend you continue the detoxification program for an additional period of time to enhance your results. Your health care provider may utilize additional nutritional supplementation based on their assessment and may recommend further testing to uncover any hidden gastrointestinal (GI) challenges.

Click the link below to sign up for the patient handbook and guide.
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https://corerestoredetox.com/

Why do we check your homocysteine level?
Homocysteine is an amino acid produced as a byproduct of our body's normal metabolic processes. To break it down and remove it, our bodies rely on essential nutrients such as folate, vitamin B6, and vitamin B12. A deficiency in any of these vitamins can lead to elevated homocysteine levels in the blood.¹
This is significant because high homocysteine levels can irritate blood vessels and increase the risk of heart disease. Elevated homocysteine may also exacerbate high blood pressure and contribute to inflammation throughout the body,² further increasing the risk of heart attacks and strokes.
To help maintain healthy homocysteine levels, it's important to consume foods rich in folate, vitamin B6, and vitamin B12. Make sure to get your yearly physical checkup with bloodwork to monitor your homocysteine levels and determine whether supplementation is necessary to protect your heart.

Foods that Contain Homocysteine-Lowering Vitamins
Folate: avocado, broccoli, eggs,  Leafy green vegetables
Vitamin B6: avocado, banana, chickpeas, spinach
Vitamin B12: salmon, tuna, eggs, Lean beef

By Caroline Berry, PA-S
UTHSCSA Graduating December 2025

SOURCES
1.Kumar A, Palfrey HA, Pathak R, Kadowitz PJ, Gettys TW, Murthy SN. The metabolism and significance of homocysteine in nutrition and health. Nutr Metab. 2017;14:78. doi:10.1186/s12986-017-0233-z
2. Ganguly P, Alam SF. Role of homocysteine in the development of cardiovascular disease. Nutr J. 2015;14:6. doi:10.1186/1475-2891-14-6

Are allergies getting the best of you this season? Natural D-Hist® is another fantastic option we carry at Deerwood Family Practice to help combat pesky allergy symptoms. D-Hist® is a targeted blend of flavonoids, antioxidants, proteolytic enzymes, and botanicals designed to support your immune system and combat symptoms caused by common environmental allergens and irritants. 
Quercetin has many amazing qualities but one trait that really shines in this product is how it helps to shut down the release of histamine. Histamine is a chemical released by your body that causes itchy watery eyes, sneezing, stuffy nose, post nasal drainage, and other allergy symptoms. 
D-Hist® also has Stinging Nettle Leaf (Urtica dioica). This plant has shown to calm and balance an overactive immune response especially in the  airway and nasal passages. It works with Quercetin to control your body’s histamine release, reducing allergy symptoms. 
Bromelain is another great component found in D-Hist®. This plant enzyme found in pineapples helps supercharge Quercetin more effectively and supports mucosal health. It helps break down large protein complexes and eases seasonal allergy discomfort. 
This combination promotes healthy nasal and sinus passages for those with high histamine levels and respiratory irritation. Additionally, Natural D-Hist® can be taken as needed or daily for year-round allergy relief, alongside other over-the-counter medications.
We also offer a safe and effective option for children! 
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SOURCES
 1.  Della Loggia R, Ragazzi E, Tubaro A, et al. Anti-inflammatory activity of benzopyrones that are inhibitors of cyco- and lipo-oxygenase. Pharmacol Res Commun. 1988;20:91-94. 
2. Kim HP, Mani I, Iversen L, Ziboh VA. Effects of naturally-occurring flavonoids and bioflavonoids on epidermal cyclooxygenase and lipoxygenase from guinea pigs. Prostagladins Leukot Essent Fatty Acids. 1998; 58:17-24. 
3. Otsuka H, Inaba M, Fujikura T, Kunitomo M. Histochemical and functional characteristics of metachromatic cells in the nasal epithelium in allergic rhinitis: studies of nasal scrapings and their dispersed cells. J Allergy Clin Immunol. 1995 ;96(4):528-36.
4. Mittman P. Randomized, double-blind study of freeze-dried Urtica dioica in the treatment of allergic rhinitis. Planta Med. 1990; 56:44-47. 
5. Obertreis, B. et al. Anti-inflammatory effect of Urtica dioica folia extract in comparison to caffeic malic acid. Arzneimittelforschung. 1996; 46(1): 52-56. 
6. Shoskes DA, Zeitlin SI, Shahed A, Rajfer J. Quercetin in men with category III chronic prostatitis: a preliminary prospective, double-blind, placebo-controlled trial. Urology. 1999; 54(6): 960-3. 
7. Cichoke AJ. The Complete Book of Enzyme Therapy. (1999). Garden City Park, NY: Avery Publishing Group.  

Addressing the Rise of Metabolic Dysfunction-Associated Steatotic Liver Disease: How Deerwood Family Practice Can Help

The rapid increase in obesity and metabolic syndrome across the U.S. is raising serious concerns about a condition known as Metabolic Dysfunction-Associated Steatotic Liver Disease (MASLD), previously known as Non-Alcoholic Fatty Liver Disease (NAFLD). This liver condition, which is not related to alcohol consumption, often presents with no symptoms in its early stages. As MASLD advances, it can lead to Metabolic Dysfunction-Associated Steatohepatitis (MASH), where harmful fat accumulates in the liver, potentially progressing to severe outcomes like fibrosis, cirrhosis, and hepatocellular carcinoma (HCC) if left untreated.
With an estimated 100 million Americans currently living with MASLD and about 6.6 million at risk of developing liver fibrosis, it’s alarming to note that fewer than 5% of MASLD cases are accurately diagnosed. Early detection and management are crucial to prevent severe liver damage and improve patient outcomes.
At Deerwood Family Practice, we are committed to advancing liver health through innovative diagnostics. We are proud to offer the OWLiver® panel, a cutting-edge, non-invasive test designed to assess the stages of MASLD with precision. Unlike traditional tests that focus solely on abnormal liver enzymes, the OWLiver® panel evaluates 28 different liver metabolites and biomarkers. This comprehensive approach provides detailed insights into the amount of fat, level of inflammation, and extent of fibrosis in the liver.
The results include the MASEF Score, a highly specific, metabolomics-driven indicator that helps identify individuals at risk for MASH. Based on your unique results, we provide a range of tailored interventions to support liver health and promote natural detoxification. Our team is continually updating our knowledge through advanced education seminars on fatty liver disease to better serve our patients.

Sources:

Barr J, Caballería J, Martínez-Arranz I, Domínguez-Díez A, Alonso C, Muntané J, Pérez-Cormenzana M, García-Monzón C, Mayo R, Martín-Duce A, Romero-Gómez M, Lo Iacono O, Tordjman J, Andrade RJ, Pérez-Carreras M, Le Marchand-Brustel Y, Tran A, Fernández-Escalante C, Arévalo E, García-Unzueta M, Clement K, Crespo J, Gual P, Gómez-Fleitas M, Martínez-Chantar ML, Castro A, Lu SC, Vázquez-Chantada M, Mato JM. Obesity-dependent metabolic signatures associated with nonalcoholic fatty liver disease progression. J Proteome Res. 2012 Apr 6;11(4):2521-32. doi: 10.1021/pr201223p. Epub 2012 Mar 15. PMID: 22364559; PMCID: PMC3321123.

Mayo R, Crespo J, Martínez-Arranz I, Banales JM, Arias M, Mincholé I, Aller de la Fuente R, Jimenez-Agüero R, Alonso C, de Luis DA, Vitek L, Stritesky J, Caballería J, Romero-Gómez M, Martín-Duce A, Mugüerza Huguet JM, Busteros-Moraza JI, Idowu MO, Castro A, Martínez-Chantar ML, Ortiz P, Bruha R, Lu SC, Bedossa P, Noureddin M, Sanyal AJ, Mato JM. Metabolomic-based noninvasive serum test to diagnose nonalcoholic steatohepatitis: Results from discovery and validation cohorts. Hepatol Commun. 2018 May 4;2(7):807-820. doi: 10.1002/hep4.1188. PMID: 30027139; PMCID: PMC6049064.

. Rinella ME, Neuschwander-Tetri BA, Siddiqui MS, et al. AASLD Practice Guidance on the clinical assessment and management of nonalcoholic fatty liver disease. Hepatology. 2023;77(5):1797-1835. doi: 10.1097/ HEP.0000000000000323

Bril F, McPhaul MJ, Kalavalapalli S, et al. Intact fasting insulin identifies nonalcoholic fatty liver disease in patients without diabetes. J Clin Endocrinol Metab. 2021;106(11):4360-e4371. doi: 10.1210/clinem/dgab417

Sarkar M, Yates K, Suzuki A, et al. Low testosterone is associated with nonalcoholic steatohepatitis and fibrosis severity in men. Clin Gastroenterol Hepatol. 2021;19(2):400-402.2. doi: 10.1016/j.cgh.2019.11.053

Kizivat T, Maric I, Mudri D, Curcic IB, Primorac D, Smolic M. Hypothyroidism and nonalcoholic fatty liver disease: pathophysiological associations and therapeutic implications. J Clin Transl Hepatol. 2020;8(3):347-
353. doi: 10.14218/JCTH.2020.00027